FcεRI-stimulated Ca2+-dependent secretion from rat basophilic leukemia (RBL-2H3) cells permeabilized with Staphylococcal α-toxin: FcεRI-operated signals are not mimicked by the actions of GTPγS
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference39 articles.
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3. The rise in concentration of free Ca2+ and of pH provides sequential, synergistic signals for secretion in antigenstimulated rat basophilic leukemia (RBL-2H3) cells;Ali;J. Immun.,1989
4. A role for calpactin in calcium-dependent exocytosis in adrenal chromaffin cells;Ali;Nature,1989
5. Characterization of hormone and protein release from α-toxin-permeabilized chromaffin cells in primary culture;Bader;J. biol. Chem.,1986
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1. Carbachol-Induced Membrane Ruffling and Its Desensitization in Rat Basophilic Leukemia (RBL-2H3) Cells Transfected with m3 Muscarinic Acetylcholine Receptors;Biological and Pharmaceutical Bulletin;2004
2. Carbachol-Induced Secretion and Homologous Desensitization in Rat Basophilic Leukemia (RBL-2H3) Cells Transfected With Human m2 Muscarinic Acetylcholine Receptors;Journal of Pharmacological Sciences;2004
3. Regulation of exocytosis by the small GTP-binding protein rho in rat basophilic leukemia (RBL-2H3) cells;General Pharmacology: The Vascular System;1995-11
4. Carbachol-induced desensitization of rat basophilic leukemia (RBL-2H3) cells transfected with human m3 muscarinic acetylcholine receptors;General Pharmacology: The Vascular System;1995-09
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