Mast Cell Stabilization Decreases Cardiomyocyte and LV Function in Dogs With Isolated Mitral Regurgitation-Reference-Cited by-同舟云学术

Mast Cell Stabilization Decreases Cardiomyocyte and LV Function in Dogs With Isolated Mitral Regurgitation

Published:2010-09 Issue:9 Volume:16 Page:769-776
ISSN:1071-9164
Container-title:Journal of Cardiac Failure
language:en
Short-container-title:Journal of Cardiac Failure

Author:

Pat Betty,Killingsworth Cheryl,Chen Yuanwen,Gladden James D.,Walcott Greg,Powell Pamela C.,Denney Thomas,Gupta Himanshu,Desai Ravi,Tillson Michael,Dillon A. Ray,Dell'Italia Louis J.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference26 articles.

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3. Volume overload cardiac hypertrophy is unaffected by ACE inhibitor treatment in the dog;Dell'Italia;Am J Physiol,1997

4. Cardiac mast cell and chymase mediated matrix metalloproteinase activity and left ventricular remodeling in mitral regurgitation in the dog;Stewart;J Mol Cell Cardiol,2003

5. Left ventricular eccentric remodeling and matrix loss are mediated by bradykinin and precede cardiomyocyte elongation in rats with volume overload;Ryan;J Am Coll Cardiol,2007

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2. Intracrine angiotensin II functions originate from noncanonical pathways in the human heart;American Journal of Physiology-Heart and Circulatory Physiology;2016-08-01

3. The Multiple Mechanistic Faces of a Pure Volume Overload: Implications for Therapy;The American Journal of the Medical Sciences;2014-10

4. Role of Chymase in Matrix and Myocardial Remodeling Due to Mitral Regurgitation: Implications for Therapy;Cardiac Remodeling;2013

5. Cardiac Adaptation to Volume Overload;Cardiac Adaptations;2012-10-12