CRISPR/Cas9 Mediated Disruption of the Swedish APP Allele as a Therapeutic Approach for Early-Onset Alzheimer’s Disease-Reference-Cited by-同舟云学术

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CRISPR/Cas9 Mediated Disruption of the Swedish APP Allele as a Therapeutic Approach for Early-Onset Alzheimer’s Disease

Published:2018-06 Issue: Volume:11 Page:429-440
ISSN:2162-2531
Container-title:Molecular Therapy - Nucleic Acids
language:en
Short-container-title:Molecular Therapy - Nucleic Acids

Author:

György Bence,Lööv Camilla,Zaborowski Mikołaj P.,Takeda Shuko,Kleinstiver Benjamin P.,Commins Caitlin,Kastanenka Ksenia,Mu Dakai,Volak Adrienn,Giedraitis Vilmantas,Lannfelt Lars,Maguire Casey A.,Joung J. Keith,Hyman Bradley T.,Breakefield Xandra O.,Ingelsson Martin

Funder

Desmond and Ann Heathwood Massachusetts General Hospital Research Scholar

Edward R. and Anne G. Lefler Center Postdoctoral Fellow

Swedish Brain Foundation

Kosciuszko Foundation

Banting

Charles A. King Trust Postdoctoral Fellowships

The Marianne and Marcus Wallenberg Foundation

NIH Common Fund

NIH

Maximizing Investigators’ Research Award

NCRR

Publisher

Elsevier BV

Subject

Drug Discovery,Molecular Medicine

Reference37 articles.

1. Systematic meta-analyses of Alzheimer disease genetic association studies: the AlzGene database;Bertram;Nat. Genet.,2007

2. The genetics of Alzheimer’s disease;Bertram;Prog. Mol. Biol. Transl. Sci.,2012

3. A pathogenic mutation for probable Alzheimer’s disease in the APP gene at the N-terminus of beta-amyloid;Mullan;Nat. Genet.,1992

4. Excessive production of amyloid beta-protein by peripheral cells of symptomatic and presymptomatic patients carrying the Swedish familial Alzheimer disease mutation;Citron;Proc. Natl. Acad. Sci. USA,1994

5. Increased beta-amyloid release and levels of amyloid precursor protein (APP) in fibroblast cell lines from family members with the Swedish Alzheimer’s disease APP670/671 mutation;Johnston;FEBS Lett.,1994

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