Tat-haFGF 14–154 Upregulates ADAM10 to Attenuate the Alzheimer Phenotype of APP/PS1 Mice through the PI3K-CREB-IRE1α/XBP1 Pathway-Reference-Cited by-同舟云学术

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Tat-haFGF 14–154 Upregulates ADAM10 to Attenuate the Alzheimer Phenotype of APP/PS1 Mice through the PI3K-CREB-IRE1α/XBP1 Pathway

Published:2017-06 Issue: Volume:7 Page:439-452
ISSN:2162-2531
Container-title:Molecular Therapy - Nucleic Acids
language:en
Short-container-title:Molecular Therapy - Nucleic Acids

Author:

Meng Tian,Cao Qin,Lei Peng,Bush Ashley I.,Xiang Qi,Su Zhijian,He Xiang,Rogers Jack T.,Chiu Ing-Ming,Zhang Qihao,Huang Yadong

Funder

National Key New Drug Creation of China

National Natural Science Foundation of China

Science & Technology Major/Planning Project of Guangdong Province

Natural Science Foundation of Guangdong Province

Science & Technology Plan Project of Guangzhou

Publisher

Elsevier BV

Subject

Drug Discovery,Molecular Medicine

Reference64 articles.

1. In vivo positron emission tomographic imaging of glial responses to amyloid-beta and tau pathologies in mouse models of Alzheimer’s disease and related disorders;Maeda;J. Neurosci.,2011

2. Brain amyloid-β oligomers in ageing and Alzheimer’s disease;Lesné;Brain,2013

3. Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer’s amyloid beta-peptide;Haass;Nat. Rev. Mol. Cell Biol.,2007

4. Activation of peroxisome proliferator-activated receptor α stimulates ADAM10-mediated proteolysis of APP;Corbett;Proc. Natl. Acad. Sci. USA,2015

5. Soluble amyloid precursor protein-α modulates β-secretase activity and amyloid-β generation;Obregon;Nat. Commun.,2012

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