The α-isoform of class II phosphoinositide 3-kinase is more effectively activated by insulin receptors than IGF receptors, and activation requires receptor NPEY motifs-Reference-Cited by-同舟云学术

The α-isoform of class II phosphoinositide 3-kinase is more effectively activated by insulin receptors than IGF receptors, and activation requires receptor NPEY motifs

Published:1999-11-01 Issue:3 Volume:460 Page:423-426
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Ursø B.,Brown R.A.,O'Rahilly S.,Shepherd P.R.,Siddle K.

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(99)01388-5/fullpdf

Reference15 articles.

1. Phosphoinositide 3-kinase: the key switch mechanism in insulin signalling

2. Phosphoinositide 3-kinases: A conserved family of signal transducers

3. Insulin Activates the α Isoform of Class II Phosphoinositide 3-Kinase

4. The signaling potential of the receptors for insulin and insulin-like growth factor I (IGF-I) in 3t3-l1 adipocytes: Comparison of glucose transport activity, induction of oncogenec-fos, glucose transporter mRNA, and DNA-synthesis

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