Growth factor protection against cytokine-induced apoptosis in neonatal rat islets of Langerhans: role of Fas
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(98)01051-5/fullpdf
Reference22 articles.
1. Inhibition of insulin secretion by interleukin-1β and tumour necrosis factor-α via an L-arginine-dependent nitric oxide generating mechanism
2. Superoxide, nitric oxide, peroxynitrite and cytokine combinations all cause functional impairment and morphological changes in rat islets of Langerhans and insulin secreting cell lines, but dictate cell death by different mechanisms
3. Human islets of Langerhans express Fas ligand and undergo apoptosis in response to interleukin-1beta and Fas ligation.
4. Cytotoxic Effects of Cytokines on Human Pancreatic Islet Cells in Monolayer Culture*
5. Interleukin-1β-Induced Nitric Oxide Production Activates Apoptosis in Pancreatic RINm5F Cells
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3. The Contribution of Reg Family Proteins to Cell Growth and Survival in Pancreatic Islets;Islets of Langerhans;2014-04-11
4. Regulation of Pancreatic Islet Beta-Cell Mass by Growth Factor and Hormone Signaling;Progress in Molecular Biology and Translational Science;2014
5. The Contribution of Reg Family Proteins to Cell Growth and Survival in Pancreatic Islets;Islets of Langerhans, 2. ed.;2014
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