Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(97)00869-7/fullpdf
Reference24 articles.
1. Myotonic Dystrophy Mutation: an Unstable CTG Repeat in the 3′ Untranslated region of the Gene
2. An Unstable Triplet Repeat in a Gene Related to Myotonic Muscular Dystrophy
3. Molecular basis of myotonic dystrophy: Expansion of a trinucleotide (CTG) repeat at the 3′ end of a transcript encoding a protein kinase family member
4. Modulation of skeletal muscle sodium channels by human myotonin protein kinase.
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1. Sodium Channel Inhibitors Reduce DMPK mRNA and Protein;Clinical and Translational Science;2015-05-22
2. Myotonic dystrophy type 1 mimics and exacerbates Brugada phenotype induced by Nav1.5 sodium channel loss-of-function mutation;Heart Rhythm;2014-08
3. Myotonic dystrophy type 1 (DM1): A triplet repeat expansion disorder;Gene;2013-06
4. About half of the late sodium current in cardiac myocytes from dog ventricle is due to non-cardiac-type Na+ channels;Journal of Molecular and Cellular Cardiology;2012-11
5. Neuromuscular excitability properties in myotonic dystrophy type 1;Clinical Neurophysiology;2007-11
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