Electrical activity regulates AChR gene expression via JNK, PKCζ and Sp1 in skeletal chick muscle
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(00)02311-5/fullpdf
Reference38 articles.
1. Acetylcholine receptor gene expression at the developing neuromuscular junction.
2. Regulation of muscle AChR α subunit gene expression by electrical activity: Involvement of protein kinase C and Ca 2+
3. Phorbol esters inhibit the activity of the chicken acetylcholine receptor alpha-subunit gene promoter. Role of myogenic regulators
4. Depolarization-transcription signals in skeletal muscle use calcium flux through L channels, but bypass the sarcoplasmic reticulum
5. Calcitonin gene-related peptide and muscle activity regulate acetylcholine receptor alpha-subunit mRNA levels by distinct intracellular pathways.
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3. Depolarization of Skeletal Muscle Cells induces Phosphorylation of cAMP Response Element Binding Protein via Calcium and Protein Kinase Cα;Journal of Biological Chemistry;2004-09
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