In vitro DNA-binding properties of VirB, theShigella flexnerivirulence regulatory protein
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(03)00524-6/fullpdf
Reference33 articles.
1. IpaB of Shigella flexneri causes entry into epithelial cells and escape from the phagocytic vacuole.
2. Shigella-induced Apoptosis Is Dependent on Caspase-1 Which Binds to IpaB
3. MxiD, an outer membrane protein necessary for the secretion of the Shigella flexneri Ipa invasins
4. Identification of icsA, a plasmid locus of Shigella flexneri that governs bacterial intra- and intercellular spread through interaction with F-actin.
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1. The virulence regulator VirB from Shigella flexneri uses a CTP-dependent switch mechanism to activate gene expression;Nature Communications;2024-01-05
2. VirB, a transcriptional activator of virulence in Shigella flexneri, uses CTP as a cofactor;Communications Biology;2023-11-25
3. VirB, a key transcriptional regulator of Shigella virulence, requires a CTP ligand for its regulatory activities;mBio;2023-10-31
4. The virulence regulator VirB fromShigella flexneriuses a CTP-dependent switch mechanism to activate gene expression;2023-06-01
5. VirB, a transcriptional activator of virulence inShigella flexneri, uses CTP as a cofactor;2023-05-19
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