Increasing cAMP antagonizes hypertrophic response to angiotensin II without affecting Ras and MAP kinase activation in vascular smooth muscle cells
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(96)01145-3/fullpdf
Reference23 articles.
1. Angiotensin II induces hypertrophy, not hyperplasia, of cultured rat aortic smooth muscle cells.
2. Angiotensin II-stimulated protein synthesis in cultured vascular smooth muscle cells.
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4. Identification of an Essential Signaling Cascade for Mitogen-activated Protein Kinase Activation by Angiotensin II in Cultured Rat Vascular Smooth Muscle Cells
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1. Ligand-activated PPARδ inhibits angiotensin II-stimulated hypertrophy of vascular smooth muscle cells by targeting ROS;PLOS ONE;2019-01-08
2. cAMP attenuates angiotensin-II-induced Egr-1 expression via PKA-dependent signaling pathway in vascular smooth muscle cells;Canadian Journal of Physiology and Pharmacology;2017-08
3. Evidence for prostacyclin and cAMP upregulation by bradykinin and insulin-like growth factor 1 in vascular smooth muscle cells;Journal of Receptors and Signal Transduction;2010-01-18
4. Key role of α1β1-integrin in the activation of PI3-kinase-Akt by flow (shear stress) in resistance arteries;American Journal of Physiology-Heart and Circulatory Physiology;2008-04
5. Participation of Reactive Oxygen Intermediates in the Angiotensin II-Activated Signaling Pathways in Vascular Smooth Muscle Cells;Annals of the New York Academy of Sciences;2006-01-25
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