Alternative splicing within the TGF-β type I receptor gene (ALK-5) generates two major functional isoforms in vascular smooth muscle cells
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(00)01132-7/fullpdf
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1. Soluble Transforming Growth Factor-β Type II Receptor Inhibits Negative Remodeling, Fibroblast Transdifferentiation, and Intimal Lesion Formation But Not Endothelial Growth
2. Genomic instability in the type II TGF-beta1 receptor gene in atherosclerotic and restenotic vascular cells.
3. Transforming growth factor-beta 1 gene activation and growth of smooth muscle from hypertensive rats.
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1. Transcriptome Bioinformatical Analysis of Vertebrate Stages of Schistosoma japonicum Reveals Alternative Splicing Events;PLOS ONE;2015-09-25
2. TGF-β and BMPR-II pharmacology—implications for pulmonary vascular diseases;Current Opinion in Pharmacology;2009-06
3. TGF-β1 Induces Human Bronchial Epithelial Cell-to-Mesenchymal Transition in Vitro;Lung;2009-02-28
4. Expression and function of alternative splice variants of the mouse TGF-β type I receptor;Cell Biology International;2008-07
5. Alternative splicing of TGF-betas and their high-affinity receptors TβRI, TβRII and TβRIII (betaglycan) reveal new variants in human prostatic cells;BMC Genomics;2007
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