Interleukin-6 reduces NMDAR-mediated cytosolic Ca2+ overload and neuronal death via JAK/CaN signaling-Reference-Cited by-同舟云学术

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Interleukin-6 reduces NMDAR-mediated cytosolic Ca2+ overload and neuronal death via JAK/CaN signaling

Published:2015-09 Issue:3 Volume:58 Page:286-295
ISSN:0143-4160
Container-title:Cell Calcium
language:en
Short-container-title:Cell Calcium

Author:

Ma Song-Hua,Zhuang Qian-Xing,Shen Wei-Xing,Peng Yu-Ping,Qiu Yi-Hua

Funder

National Natural Science Foundation of China

Foundation of Jiangsu Higher Education Committee

Nantong Applied Research Program

Priority Academic Program Development (PAPD) of Jiangsu Higher Education Institutions

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference62 articles.

1. Chronic interleukin-6 exposure alters metabotropic glutamate receptor-mediated calcium signaling in cerebellar Purkinje neurons;Nelson;Eur. J. Neurosci.,2004

2. Bax regulates neuronal Ca2+ homeostasis;D’Orsi;J. Neurosci.,2015

3. Mitochondrial sensitivity and altered calcium handling underlie enhanced NMDA-induced apoptosis in YAC128 model of Huntington's disease;Fernandes;J. Neurosci.,2007

4. Glutamate receptor ion channels: structure, regulation, and function;Traynelis;Pharmacol. Rev.,2010

5. NMDA receptor subunit expression in the supraoptic nucleus of adult rats: dominance of NR2B and NR2D;Doherty;Brain Res.,2011

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