Inositol-1,4,5-triphosphate receptors mediate activity-induced synaptic Ca2+ signals in muscle fibers and Ca2+ overload in slow-channel syndrome

Author:

Zayas Roberto,Groshong Jason S.,Gomez Christopher M.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference68 articles.

1. Junctional membrane structure and store operated calcium entry in muscle cells;Ma;Front. Biosci.,2003

2. Dihydropyridine receptors as voltage sensors for a depolarization-evoked, IP3R-mediated, slow calcium signal in skeletal muscle cells;Araya;J. Gen. Physiol.,2003

3. Novel delta subunit mutation in slow-channel syndrome causes severe weakness by novel mechanisms;Gomez;Ann. Neurol.,2002

4. Congenital disorders of neuromuscular transmission;Engel;Semin. Neurol.,1990

5. Congenital myasthenic syndrome attributed to a mutation of the epsilon subunit of the acetylcholine receptor (AChR);Engel;Neurology,1992

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