Bisphenol A Workshop of the German Federal Environment Agency – March 30–31, 2009
Author:
Publisher
Elsevier BV
Subject
Public Health, Environmental and Occupational Health
Reference20 articles.
1. Low doses of bisphenol A promote human seminoma cell proliferation by activating PKA and PKG via a membrane G-protein-coupled estrogen receptor;Bouskine;Environ. Health Perspect.,2009
2. Exposure to bisphenol A and other phenols in neonatal intensive care unit premature infants;Calafat;Environ. Health Perspect.,2009
3. NTP-CERHR expert panel report on the reproductive and developmental toxicity of bisphenol A;Charpin;Birth Defects Res. B – Dev. Reprod. Toxicol.,2008
4. Predicting plasma concentrations of bisphenol A in children younger than 2 years of age after typical feeding schedules, using a physiologically based toxicokinetic model;Edginton;Environ. Health Perspect.,2009
5. Opinion of the scientific panel on food additives, flavourings, processing aids and materials in contact with food on a request from the Commission related to Bisphenol A;EFSA;EFSA J.,2006
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1. The Conflict between Regulatory Agencies over the 20,000-Fold Lowering of the Tolerable Daily Intake (TDI) for Bisphenol A (BPA) by the European Food Safety Authority (EFSA);Environmental Health Perspectives;2024-04
2. Precise Yet Uncertain: Broadening Understandings of Uncertainty and Policy in the BPA Controversy;Risk Analysis;2021-12
3. Perinatal exposure to Bisphenol S (BPS) promotes obesity development by interfering with lipid and glucose metabolism in male mouse offspring;Environmental Research;2019-06
4. Flaws in design, execution and interpretation limit CLARITY‐BPA’s value for risk assessments of bisphenol A;Basic & Clinical Pharmacology & Toxicology;2019-02-15
5. Why did researchers not use realistic doses in animal studies of bisphenol A?;Archives of Toxicology;2016-09-08
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