ADAMTS18 deficiency leads to preputial gland hypoplasia and fibrosis in male mice
Author:
Funder
Natural Science Foundation of Shanghai
National Natural Science Foundation of China
Publisher
Elsevier BV
Subject
Developmental Biology,Endocrinology,Animal Science and Zoology
Reference23 articles.
1. A new biological marker candidate in female reproductive system diseases: matrix metalloproteinase with thrombospondin motifs (ADAMTS);Demircan;J Turk Ger Gynecol Assoc,2014
2. Transgenic mice with inactive alleles for procollagen N-proteinase (ADAMTS-2) develop fragile skin and male sterility;Li;Biochem J,2001
3. Male mice lacking ADAMTS-16 are fertile but exhibit testes of reduced weight;Livermore;Sci Rep,2019
4. Epigenetic identification of ADAMTS18 as a novel 16q23.1 tumor suppressor frequently silenced in esophageal, nasopharyngeal and multiple other carcinomas;in;Oncogene,2007
5. Identification of ADAMTS18 as a gene mutated in Knobloch syndrome;Aldahmesh;J Med Genet,2011
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1. The progression of obstructive renal fibrosis in rats is regulated by ADAMTS18 gene methylation in the embryonic kidney through the AKT/Notch pathway;Journal of Biochemical and Molecular Toxicology;2023-12-26
2. Secreted protease ADAMTS18 in development and disease;Gene;2023-03
3. Comparative Transcriptome Profiles of Human HaCaT Cells in Response to Gynostemma pentaphyllum Extracts Obtained Using Three Independent Methods by RNA Sequencing;Life;2023-02-02
4. ADAMTS18 regulates early branching morphogenesis of lacrimal gland and has a significant association with the risk of dry eye in mice;Experimental Eye Research;2022-05
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