Long‐term basal forebrain cholinergic‐rich graftsderived from trisomy 16 mice do not develop β ‐AMYLOID pathology and neurodegeneration butdemonstrate neuroinflammatory responses-Reference-Cited by-同舟云学术

Long‐term basal forebrain cholinergic‐rich graftsderived from trisomy 16 mice do not develop β ‐AMYLOID pathology and neurodegeneration butdemonstrate neuroinflammatory responses

Published:1998-11 Issue:7-8 Volume:16 Page:763-775
ISSN:0736-5748
Container-title:International Journal of Developmental Neuroscience
language:en
Short-container-title:Int. j. dev. neurosci.

Author:

Stahl Tobias¹,Goldammer Axel¹,Luschekina Elena²,Beck Mike¹,Schliebs Reinhard¹,Bigl Volker¹

Affiliation:

1. Paul Flechsig Institute for Brain ResearchDepartment of NeurochemistryUniversity of Leipzig Medical FacultyJahnallee 59D‐04109LeipzigGermany

2. Institute of Higher Nervous ActivityRussian Academy of SciencesMoscowRussia

Funder

Sächsisches Ministerium für Umwelt und Landesentwicklung

Schering AG and the Bundesministerium für Bildung und Forschung

Publisher

Wiley

Subject

Developmental Biology,Developmental Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1016/S0736-5748%2898%2900085-9

Reference42 articles.

1. Estimation of nuclear population from microtome sections

2. Glutamate as a hippocampal neuron survival factor: an inherited defect in the trisomy 16 mouse.

3. Neuropeptide Y immunoreactive neurons in murine trisomy 16 cortical cultures

4. Comparative mapping of DNA markers from the familial Alzheimer disease and Down syndrome regions of human chromosome 21 to mouse chromosomes 16 and 17.

5. Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction

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