Molecular cloning of Chinese hamster ceramide glucosyltransferase and its enhanced expression in peroxisome-defective mutant Z65 cells
Author:
Publisher
Elsevier BV
Subject
Molecular Biology,Biochemistry,Biophysics
Reference31 articles.
1. Accumulation of glycolipids in mutant Chinese hamster ovary cells (Z65) with defective peroxisomal assembly and comparison of the metabolic rate of glycosphingolipids between Z65 cells and wild-type CHO-K1 cells
2. A Human Gene Responsible for Zellweger Syndrome That Affects Peroxisome Assembly
3. Isolation and characterization of Chinese hamster ovary cell mutants defective in assembly of peroxisomes.
4. Cerebro-hepato-renal syndrome of Zellweger: An inherited disorder of neuronal migration
5. The mechanism of arrest of neuronal migration in the Zellweger malformation: An hypothesis based upon cytoarchitectonic analysis
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Dissociation of ERK signalling inhibition from the anti-amyloidogenic action of synthetic ceramide analogues;Clinical Science;2012-01-16
2. Alterations in the molecular species of plasmalogen phospholipids and glycolipids due to peroxisomal dysfunction in Chinese hamster ovary-mutant Z65 cells by FABMS method;Journal of Chromatography B;2007-06
3. Increase of ceramide monohexoside and dipalmitoyl glycerophospholipids in the brain of Zellweger syndrome;Neuroscience Letters;2007-05
4. Alterations of fatty acid metabolism and membrane fluidity in peroxisome-defective mutant ZP102 cells;Lipids;2004-01
5. Ceramide glucosyltransferase;Springer Handbook of Enzymes
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