SMUG1 regulates fat homeostasis leading to a fatty liver phenotype in mice-Reference-Cited by-同舟云学术

SMUG1 regulates fat homeostasis leading to a fatty liver phenotype in mice

Published:2022-12 Issue: Volume:120 Page:103410
ISSN:1568-7864
Container-title:DNA Repair
language:en
Short-container-title:DNA Repair

Author:

Carracedo Sergio,Lirussi Lisa,Alsøe Lene,Segers Filip,Wang Changliang,Bartosova Zdenka,Bohov Pavol,Tekin Nuriye B.,Kong Xiang Yi,Esbensen Q. Ying,Chen Liang,Wennerström Anna,Kroustallaki Penelope,Ceolotto Deborah,Tönjes Anke,Berge Rolf Kristian,Bruheim Per,Wong Garry,Böttcher Yvonne,Halvorsen Bente,Nilsen Hilde

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference69 articles.

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2. Uracil accumulation and mutagenesis dominated by cytosine deamination in CpG dinucleotides in mice lacking UNG and SMUG1;Alsoe;Sci. Rep.,2017

3. hUNG2 is the major repair enzyme for removal of uracil from U:A matches, U:G mismatches, and U in single-stranded DNA, with hSMUG1 as a broad specificity backup;Kavli;J. Biol. Chem.,2002

4. Germline ablation of SMUG1 DNA glycosylase causes loss of 5-hydroxymethyluracil- and UNG-backup uracil-excision activities and increases cancer predisposition of Ung-/-Msh2-/- mice;Kemmerich;Nucleic Acids Res.,2012

5. Excision of deaminated cytosine from the vertebrate genome: role of the SMUG1 uracil-DNA glycosylase;Nilsen;EMBO J.,2001

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