Mouse models of DNA mismatch repair in cancer research
Author:
Funder
NIH
Feinberg Family Foundation donation
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference64 articles.
1. MSH2 deficient mice are viable and susceptible to lymphoid tumours;Reitmair;Nat. Genet.,1995
2. Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancer;de Wind;Cell,1995
3. Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity;Smits;Genes Chromosom. Cancer,2000
4. Mutation in the mismatch repair gene Msh6 causes cancer susceptibility;Edelmann;Cell,1997
5. Tumour susceptibility and spontaneous mutation in mice deficient in Mlh1, Pms1 and Pms2 DNA mismatch repair;Prolla;Nat. Genet.,1998
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