PCNA ubiquitination-independent activation of polymerase η during somatic hypermutation and DNA damage tolerance-Reference-Cited by-同舟云学术

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PCNA ubiquitination-independent activation of polymerase η during somatic hypermutation and DNA damage tolerance

Published:2011-10 Issue:10 Volume:10 Page:1051-1059
ISSN:1568-7864
Container-title:DNA Repair
language:en
Short-container-title:DNA Repair

Author:

Krijger Peter H.L.,van den Berk Paul C.M.,Wit Niek,Langerak Petra,Jansen Jacob G.,Reynaud Claude-Agnès,de Wind Niels,Jacobs Heinz

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference60 articles.

1. Molecular mechanisms of antibody somatic hypermutation;Di Noia;Annu. Rev. Biochem.,2007

2. Dependence of antibody gene diversification on uracil excision;Di Noia;J. Exp. Med.,2007

3. Mismatch recognition and uracil excision provide complementary paths to both Ig switching and the A/T-focused phase of somatic mutation;Rada;Mol. Cell,2004

4. Increased hypermutation at G and C nucleotides in immunoglobulin variable genes from mice deficient in the MSH2 mismatch repair protein;Phung;J. Exp. Med.,1998

5. Hot spot focusing of somatic hypermutation in MSH2-deficient mice suggests two stages of mutational targeting;Rada;Immunity,1998

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3. Huwe1 supports B-cell development, B-cell-dependent immunity, somatic hypermutation and class switch recombination by regulating proliferation;Frontiers in Immunology;2023-01-09

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