A SEMA3E mutant resistant to cleavage by furins (UNCL-SEMA3E) inhibits choroidal neovascularization-Reference-Cited by-同舟云学术

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A SEMA3E mutant resistant to cleavage by furins (UNCL-SEMA3E) inhibits choroidal neovascularization

Published:2016-12 Issue: Volume:153 Page:186-194
ISSN:0014-4835
Container-title:Experimental Eye Research
language:en
Short-container-title:Experimental Eye Research

Author:

Toledano Shira,Lu Huayi,Palacio Agustina,Kigel Boaz,Kessler Ofra,Allon Gilad,Barak Yoreh,Neufeld Gera,Schaal Shlomit

Funder

Israel Binational Science Foundation (BSF)

Israel Ministry of Commerce

Israel Science Foundation

Rappaport Family Institute for Research in the Medical Sciences

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology

Reference43 articles.

1. The chemorepulsive activity of secreted semaphorins is regulated by furin-dependent proteolytic processing;Adams;EMBO J.,1997

2. Proprotein convertases: “master switches” in the regulation of tumor growth and progression;Bassi;Mol. Carcinog.,2005

3. VEGFR2 (KDR/Flk1) signaling mediates axon growth in response to semaphorin 3E in the developing brain;Bellon;Neuron,2010

4. Ranibizumab versus verteporfin photodynamic therapy for neovascular age-related macular degeneration: two-year results of the ANCHOR study;Brown;Ophthalmology,2009

5. The burden of age-related macular degeneration: a value-based analysis. Curr. Opin;Brown;Ophthalmol,2006

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