RNA sequencing uncovers alterations in corneal endothelial metabolism, pump and barrier functions of Slc4a11 KO mice
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology
Reference27 articles.
1. Ophthalmic nonsteroidal anti-inflammatory drugs as a therapy for corneal dystrophies caused by SLC4A11 mutation;Alka;Invest. Ophthalmol. Vis. Sci.,2018
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3. Intestinal epithelial claudins: expression and regulation in homeostasis and inflammation;Garcia-Hernandez;Ann. N. Y. Acad. Sci.,2017
4. SLC4A11 depletion impairs NRF2 mediated antioxidant signaling and increases reactive oxygen species in human corneal endothelial cells during oxidative stress;Guha;Sci. Rep.,2017
5. Enhanced expression of SLC4A11 by tert-Butylhydroquinone is mediated by direct binding of Nrf2 to the promoter of SLC4A11;Guha;Free Radic. Biol. Med.,2021
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Investigation of the functional impact of CHED- and FECD4-associated SLC4A11 mutations in human corneal endothelial cells;PLOS ONE;2024-01-22
2. Corneal Edema in Inducible Slc4a11 Knockout Is Initiated by Mitochondrial Superoxide Induced Src Kinase Activation;Cells;2023-06-01
3. Mitochondrial ROS in Slc4a11 KO Corneal Endothelial Cells Lead to ER Stress;Frontiers in Cell and Developmental Biology;2022-04-26
4. Effect of Physiological Oxygen on Primary Human Corneal Endothelial Cell Cultures;Translational Vision Science & Technology;2022-02-22
5. The H+ Transporter SLC4A11: Roles in Metabolism, Oxidative Stress and Mitochondrial Uncoupling;Cells;2022-01-07
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