High glucose and oxidative/nitrosative stress conditions induce apoptosis in retinal endothelial cells by a caspase-independent pathway-Reference-Cited by-同舟云学术

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High glucose and oxidative/nitrosative stress conditions induce apoptosis in retinal endothelial cells by a caspase-independent pathway

Published:2009-05 Issue:5 Volume:88 Page:983-991
ISSN:0014-4835
Container-title:Experimental Eye Research
language:en
Short-container-title:Experimental Eye Research

Author:

Leal Ermelindo C.,Aveleira Célia A.,Castilho Áurea F.,Serra Andreia M.,Baptista Filipa I.,Hosoya Ken-Ichi,Forrester John V.,Ambrósio António F.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology

Reference40 articles.

1. Expression of antiapoptotic and proapoptotic molecules in diabetic retinas;Abu El-Asrar;Eye,2007

2. Mechanisms of high glucose-induced apoptosis and its relationship to diabetic complications;Allen;J. Nutr. Biochem.,2005

3. Diabetes-enhanced tumor necrosis factor-alpha production promotes apoptosis and the loss of retinal microvascular cells in type 1 and type 2 models of diabetic retinopathy;Behl;Am. J. Pathol.,2008

4. Hyperglycemia-induced reactive oxygen species toxicity to endothelial cells is dependent on paracrine mediators;Busik;Diabetes,2008

5. Studies on the pathophysiology of diabetic retinopathy. The blood–retinal barrier in diabetes;Cunha-Vaz;Diabetes,1983

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