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BetaB2-crystallin mutations associated with cataract and glaucoma leads to mitochondrial alterations in lens epithelial cells and retinal neurons

  • Published:2017-02 Issue: Volume:155 Page:85-90
  • ISSN:0014-4835
  • Container-title:Experimental Eye Research
  • language:en
  • Short-container-title:Experimental Eye Research

Author:

Dulle Jennifer E.,Rübsam Anne,Garnai Sarah J.,Pawar Hemant S.,Fort Patrice E.ORCID

Funder

NEI

international retina research foundation (IRRF) postdoctoral fellowship (J.E.D.)

Dr. Werner Jackstaedt Foundation (A.R.)

National Eye Institute

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology

Reference39 articles.

1. Glaucoma after congenital cataract surgery;Asrani;Ophthalmology,1995

2. betaB2-Crystallin promotes axonal regeneration in the injured optic nerve in adult rats;Bohm;Cell Transpl.,2015

3. Factors affecting visual outcome after surgery for bilateral congenital cataracts;Bradford;Am. J. Ophthalmol.,1994

4. Deletion mutation in an eye lens beta-crystallin. An animal model for inherited cataracts;Chambers;J. Biol. Chem.,1991

5. A missense mutation in CRYBB2 leads to progressive congenital membranous cataract by impacting the solubility and function of betaB2-crystallin;Chen;PLoS One,2013

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