Lipid-derived and other oxidative modifications of retinal proteins in a rat model of Smith-Lemli-Opitz syndrome-Reference-Cited by-同舟云学术

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Lipid-derived and other oxidative modifications of retinal proteins in a rat model of Smith-Lemli-Opitz syndrome

Published:2019-01 Issue: Volume:178 Page:247-254
ISSN:0014-4835
Container-title:Experimental Eye Research
language:en
Short-container-title:Experimental Eye Research

Author:

Kapphahn Rebecca J.,Richards Michael J.,Ferrington Deborah A.,Fliesler Steven J.^ORCID

Funder

National Institutes of Health

SUNY-University

National Center for Advancing Translational Sciences

University of Minnesota

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology

Reference62 articles.

1. Protein oxidation in aging, disease, and oxidative stress;Berlett;J. Biol. Chem.,1997

2. Protein tyrosine nitration: role in aging;Chakravarti;Curr. Aging Sci.,2017

3. Posttranslational modifications of human αA-crystallin: correlation with electrophoretic migration;Colvis;Arch. Biochem. Biophys.,2002

4. Smith-Lemli-Opitz syndrome;DeBarber;Expet Rev. Mol. Med.,2011

5. Lipoxidation adducts with peptides and proteins: deleterious modifications or signaling mechanisms?;Domingues;J. Proteomics,2013

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3. Transcriptomic Changes Associated with Loss of Cell Viability Induced by Oxysterol Treatment of a Retinal Photoreceptor-Derived Cell Line: An In Vitro Model of Smith–Lemli–Opitz Syndrome;International Journal of Molecular Sciences;2021-02-26

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