The negative regulatory Spred1 and Spred2 proteins are required for lens and eye morphogenesis
Author:
Funder
University of Sydney
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology
Reference49 articles.
1. Expression and subcellular localization of Spred proteins in mouse and human tissues;Engelhardt;Histochem. Cell Biol.,2004
2. Spred-1 negatively regulates allergen-induced airway eosinophilia and hyperresponsiveness;Inoue;J. Exp. Med.,2005
3. Distinct requirements for the Sprouty domain for functional activity of Spred proteins;King;Biochem. J.,2005
4. Fibre cell organization in crystalline lenses.;Kuszak;Exp Eye Res,2004
5. Expression and activity of the signaling molecules for mitogen-activated protein kinase pathways in human, bovine, and rat lenses;Li;Investig. Ophthalmol. Vis. Sci.,2003
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1. Conditional Ablation of Spred1 and Spred2 in the Eye Lens Negatively Impacts Its Development and Growth;Cells;2024-02-06
2. Fibroblast Growth Factor-induced lens fiber cell elongation is driven by the stepwise activity of Rho and Rac;2023-12-05
3. A possible connection between reactive oxygen species and the unfolded protein response in lens development: From insight to foresight;Frontiers in Cell and Developmental Biology;2022-09-21
4. Sprouty-related proteins with EVH1 domain (SPRED2) prevents high-glucose induced endothelial–mesenchymal transition and endothelial injury by suppressing MAPK activation;Bioengineered;2022-05-02
5. Lens fiber cell differentiation occurs independently of fibroblast growth factor receptor signaling in the absence of Pten;Developmental Biology;2020-11
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