Loss of heterozygosity of p16 correlates with minimal residual disease at the end of the induction therapy in non-high risk childhood B-cell precursor acute lymphoblastic leukemia-Reference-Cited by-同舟云学术

Loss of heterozygosity of p16 correlates with minimal residual disease at the end of the induction therapy in non-high risk childhood B-cell precursor acute lymphoblastic leukemia

Published:2002-09 Issue:9 Volume:26 Page:817-820
ISSN:0145-2126
Container-title:Leukemia Research
language:en
Short-container-title:Leukemia Research

Author:

Tutor Olga,Dı́az Miguel A,Ramı́rez Manuel,Algara Patricia,Madero Luis,Martı́nez Pedro

Publisher

Elsevier BV

Subject

Cancer Research,Oncology,Hematology

Reference17 articles.

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4. p16INK4A gene and hematological malignancies;Quesnel;Leuk. Lymphoma,1996

5. Genetic studies of childhood acute lymphoblastic leukemia with emphasis on p16, MLL and ETV6 gene abnormalities: results of St. Jude total therapy study XII;Rubnitz;Leukemia,1997

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2. Tumour suppressor genes in chemotherapeutic drug response;Bioscience Reports;2012-04-23

3. Effect of p16 on glucocorticoid response in a B-cell lymphoblast cell line;Korean Journal of Pediatrics;2010

4. Clinical significance of loss of p16 protein by immunohistochemical staining in acute lymphoblastic leukemia;Korean Journal of Pediatrics;2008

5. The co-transfection of p16INK4a and p14ARF genes into human lung cancer cell line A549 and the effects on cell growth and chemosensitivity;Colloids and Surfaces B: Biointerfaces;2005-12