Mitochondrial damage and activation of NLRP3 induced by Yersinia ruckeri infection play an important role in the occurrence of diffuse acute inflammation of channel catfish (Ictalurus punctatus)

Author:

Lu Chen-Wang,He Jie,Xiong Quan-Xin,Zhong Zhen-Dong,Yang Bin,Lian Sheng,Wang Meng-Meng,Bai Shang-Jie,Kuang Lin-Feng,Zhou Zheng-Yang,Jian Zheng-Ran,Gao Yuan-Chang,He Sheng-Yu,Wang Er-Long,Ling Fei,Xiao Shuang-Yan,Liu Ming-Zhu,Li Peng-Fei,Wang Gao-Xue,Yu Qing,Liu Tao

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Reference35 articles.

1. How location and cellular signaling combine to activate the NLRP3 inflammasome;Akbal;Cell. Mol. Immunol.,2022

2. Pathological findings in rainbow trout (Oncorhynchus mykiss Walbaum, 1792) experimentally infected with Yersinia ruckeri;Avci;Turk. J. Vet. Anim. Sci.,2005

3. Phylogeography of Yersinia ruckeri reveals effects of past evolutionary events on the current strain distribution and explains variations in the global transmission of enteric redmouth (ERM) disease;Bastardo;Front. Microbiol.,2015

4. Mitochondria as multifaceted regulators of cell death;Bock;Nat. Rev. Mol. Cell Biol.,2020

5. Differential binding of NLRP3 to non-oxidized and ox-mtDNA mediates NLRP3 inflammasome activation;Cabral;Commun. Biol.,2023

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