Enprostil, a synthetic prostaglandin E2 analogue, inhibits meal-stimulated gastric acid secretion and gastrin release in patients with duodenal ulcer
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference26 articles.
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3. The effect of 16, 16-dimethyl prostaglandin E2 on meal-stimulated gastric acid secretion and serum gastrin in duodenal ulcer patients;Ippoliti;Gastroenterology,1976
4. Comparison of methylated prostaglandin E2 analogues given orally in the inhibition of gastric responses to pentagastrin and peptone meal in man;Konturek;Gastroenterology,1976
5. Effect of synthetic 15-methyl analog of PGE2 on gastric acid and serum gastrin response to peptone meal, pentagastrin, and histamine in duodenal ulcer patients;Konturek;Dig Dis Sci,1976
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1. The effect of enprostil on duodeno-jejunal motility in man;Alimentary Pharmacology & Therapeutics;2007-03-31
2. Enprostil inhibits post-prandial gastrin release: a dose-response study;Alimentary Pharmacology & Therapeutics;2007-03-31
3. Effect of enprostil on serum gastrin and pepsinogen A and C levels in patients on long-term treatment with omeprazole;Alimentary Pharmacology & Therapeutics;2007-03-31
4. Effect of enprostil on the gastroduodenal mucosa of healthy volunteers;Alimentary Pharmacology & Therapeutics;2007-03-31
5. Anti-secretory effects and pharmacokinetics of low dose ranitidine;Alimentary Pharmacology & Therapeutics;2007-03-31
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