Effects of endotoxic shock on neuronal NOS and calcium transients in rat cardiac myocytes-Reference-Cited by-同舟云学术

Effects of endotoxic shock on neuronal NOS and calcium transients in rat cardiac myocytes

Published:2005-05 Issue:5 Volume:51 Page:409-417
ISSN:1043-6618
Container-title:Pharmacological Research
language:en
Short-container-title:Pharmacological Research

Author:

Comini Laura,Boraso Antonella,Bachetti Tiziana,Bernocchi Palmira,Pasini Evasio,Bastianon Daniela,Curello Salvatore,Terracciano Cesare Maria,Ceconi Claudio,Ferrari Roberto

Publisher

Elsevier BV

Subject

Pharmacology

Reference39 articles.

1. The cardiovascular response of normal humans to the administration of endotoxin;Suffredini;N Engl J Med,1989

2. Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock;Natanson;J Exp Med,1989

3. Pathogenetic mechanisms of septic shock;Parrillo;N Engl J Med,1993

4. Nitric oxide synthases: which, where, why and how?;Michel;J Clin Invest,1997

5. Expression of inducible nitric oxide synthase in human heart failure;Haywood;Circulation,1996

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