Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a− mice-Reference-Cited by-同舟云学术

Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a− mice

Published:2020-09 Issue: Volume:159 Page:104922
ISSN:1043-6618
Container-title:Pharmacological Research
language:en
Short-container-title:Pharmacological Research

Author:

Patin Justine,Castro Claire,Steenman Marja^ORCID,Hivonnait Agnès,Carcouët Agnès,Tessier Arnaud,Lebreton Jacques,Bihouée Audrey^ORCID,Donnart Audrey,Le Marec Hervé,Baró Isabelle,Charpentier Flavien,Derangeon Mickaël

Funder

Agence Nationale de la Recherche

Publisher

Elsevier BV

Subject

Pharmacology

Reference49 articles.

1. Common and unique mechanisms regulate fibrosis in various fibroproliferative diseases;Wynn;J. Clin. Invest.,2007

2. Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease;Gourdie;Nat. Rev. Drug Discov.,2016

3. Transforming growth factor β receptor inhibition prevents ventricular fibrosis in a mouse model of progressive cardiac conduction disease;Derangeon;Cardiovasc. Res.,2017

4. Mouse model of SCN5A-linked hereditary Lenègre’s disease: age-related conduction slowing and myocardial fibrosis;Royer;Circulation,2005

5. Impaired impulse propagation in Scn5a-knockout mice;Van Veen;Circulation,2005

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MicroRNA-452-5p regulates fibrogenesis via targeting TGF-β/SMAD4 axis in SCN5A-knockdown human cardiac fibroblasts;iScience;2024-06

2. Mannitol and hyponatremia regulate cardiac ventricular conduction in the context of sodium channel loss of function;American Journal of Physiology-Heart and Circulatory Physiology;2024-03-01

3. Brugada Syndrome: More than a Monogenic Channelopathy;Biomedicines;2023-08-18

4. Interindividual Age-Independent Differences in Human CX43 Impact Ventricular Arrhythmic Risk;Research;2023-01

5. S-Doped carbonized polymer dots inhibit early myocardial fibrosis by regulating mitochondrial function;Biomaterials Science;2023