ROS-related mitochondrial dysfunction in skeletal muscle of an ALS mouse model during the disease progression-Reference-Cited by-同舟云学术

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ROS-related mitochondrial dysfunction in skeletal muscle of an ALS mouse model during the disease progression

Published:2018-12 Issue: Volume:138 Page:25-36
ISSN:1043-6618
Container-title:Pharmacological Research
language:en
Short-container-title:Pharmacological Research

Author:

Xiao Yajuan,Karam Chehade,Yi Jianxun,Zhang Lin,Li Xuejun,Yoon Dosuk,Wang Huan,Dhakal Kamal,Ramlow Paul,Yu Tian,Mo Zhaohui,Ma Jianjie,Zhou Jingsong

Funder

NIAMS/NIH

Bank of American Victor E. Speas Foundation and ALS Association

McCown Gordon Gala Research Gift

NIH/NHLBI T32

Zunyi Medical University

Publisher

Elsevier BV

Subject

Pharmacology

Reference79 articles.

1. Incidence and lifetime risk of motor neuron disease in the United Kingdom: a population-based study;Alonso;Eur. J. Neurol.,2009

2. Molecular biology of amyotrophic lateral sclerosis: insights from genetics;Pasinelli;Nat. Rev. Neurosci.,2006

3. ALS: a disease of motor neurons and their nonneuronal neighbors;Boillee;Neuron,2006

4. Skeletal muscle is a primary target of SOD1G93A-mediated toxicity;Dobrowolny;Cell Metab.,2008

5. Defective mitochondrial dynamics is an early event in skeletal muscle of an amyotrophic lateral sclerosis mouse model;Luo;PLoS One,2013

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