Cullin-associated and neddylation-dissociated 1 protein (CAND1) governs cardiac hypertrophy and heart failure partially through regulating calcineurin degradation

Author:

Li Xingda,Zhang Yang,Zhao Yue,Zhou Yang,Han Qilong,Yang Ying,Zhang Lingmin,Shi Ling,Jin Xuexin,Zhang Ruixin,Gao Haiyu,Xue Genlong,Li Desheng,Zhang Zhi-Ren,Lu Yanjie,Yang Baofeng,Pan Zhenwei

Publisher

Elsevier BV

Subject

Pharmacology

Reference33 articles.

1. Cardiac hypertrophy: the good, the bad, and the ugly;Frey;Annu. Rev. Physiol.,2003

2. Pathological ventricular remodeling: mechanisms: part 1 of 2;Burchfield;Circulation,2013

3. Structural insights into the catalysis and regulation of E3 ubiquitin ligases;Buetow;Nat. Rev. Mol. Cell Biol.,2016

4. The ubiquitin-proteasome system: a potential therapeutic target for heart failure;Barac;J. Heart Lung Transplant. Off. Publ. Int. Soc. Heart Transplant.,2017

5. The cullin protein family;Sarikas;Genome Biol.,2011

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