Differential changes in sympathetic activity in left and right ventricles in congestive heart failure after myocardial infarction
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference22 articles.
1. Rapid activation of the sympathetic nervous system following coronary artery occlusion: relationship to infarct size, site and hemodynamic impact;Karlsberg;Cardiovasc Res,1979
2. Plasma catecholamines in acute myocardial infarction;Nadeau;Am Heart J,1979
3. Plasma norepinephrine as an indicator of sympathetic neural activity in clinical cardiology;Goldstein;Am J Cardiol,1981
4. Myocardial catecholamines and the pathophysiology of heart failure;Daly;Circulation,1990
5. Cardiac catecholamine synthesis, turnover and metabolism with isoproterenol-induced myocytolysis;Mueller;Cardiovasc Res,1971
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1. Oxidative Products of Catecholamines During Heightened Sympathetic Activity Due to Congestive Heart Failure: Possible Role of Antioxidants;International Journal of General Medicine;2024-03
2. Behavior of Hypertrophied Right Ventricle during the Development of Left Ventricular Failure Due to Myocardial Infarction;International Journal of Molecular Sciences;2024-02-23
3. Prolonged β-adrenergic stimulation disperses ryanodine receptor clusters in cardiomyocytes and has implications for heart failure;eLife;2022-08-01
4. T-tubule remodelling disturbs localized β2-adrenergic signalling in rat ventricular myocytes during the progression of heart failure;Cardiovascular Research;2017-05-13
5. Adenylyl cyclase regulation in heart failure due to myocardial infarction in rats;Naunyn-Schmiedeberg's Archives of Pharmacology;2013-11-26
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