Occupational lead exposure: Studies in two brothers showing differential susceptibility to lead

Author:

Haust H.L.,Poon H.C.,Inwood M.,O'Hea E.K.,Haines D.S.M.,Forret C.J.,Keys D.S.

Publisher

Elsevier BV

Subject

Clinical Biochemistry,General Medicine

Reference36 articles.

1. The relationship of lead poisoning to acute intermittent porphyria;Dagg;Q J Med,1964

2. Heme biosynthesis in intermittent acute porphyria: decreased hepatic conversion of porphobilinogen to porphyrins and increased delta-aminolaevulinic acid synthetase activity;Strand,1970

3. The serum porphobilinogen and hepatic porphobilinogen deaminase in normal and porphyric individuals;Miyagi;J Lab Clin Med,1971

4. Decreased red cell uroporphyrinogen I synthetase activity in intermittent acute porphyria;Strand;J Clin Invest,1972

5. δ-aminolaevulinic acid dehydratase activity in circulating red cells. A sensitive laboratory test for the detection of childhood lead poisoning;Weissberg;N Engl J Med,1971

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