The Power of the Dark Side: Huntington’s Disease Protein and p53 Form a Deadly Alliance
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference13 articles.
1. p53 Mediates Cellular Dysfunction and Behavioral Abnormalities in Huntington’s Disease
2. Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid
3. p53 is Present in Synapses Where it Mediates Mitochondrial Dysfunction and Synaptic Degeneration in Response to DNA Damage, and Oxidative and Excitotoxic Insults
4. Contribution of p53-Dependent Caspase Activation to Neuronal Cell Death Declines with Neuronal Maturation
5. Extracellular deposition of beta-amyloid upon p53-dependent neuronal cell death in transgenic mice.
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1. An RNA-targeting CRISPR–Cas13d system alleviates disease-related phenotypes in Huntington’s disease models;Nature Neuroscience;2022-12-12
2. Mitochondrial Dysfunction in Huntington’s Disease; Interplay Between HSF1, p53 and PGC-1α Transcription Factors;Frontiers in Cellular Neuroscience;2019-03-19
3. Emerging Roles of Mitochondrial Serine Protease HtrA2 in Neurodegeneration;Proteases in Physiology and Pathology;2017
4. Interactome network analysis identifies multiple caspase-6 interactors involved in the pathogenesis of HD;Human Molecular Genetics;2016-02-11
5. Involvement of the Mesocorticolimbic Dopaminergic System in Adaptive Behavior;Neuroscience and Behavioral Physiology;2011-09
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