A central mechanism of analgesia in mice and humans lacking the sodium channel NaV1.7-Reference-Cited by-同舟云学术

A central mechanism of analgesia in mice and humans lacking the sodium channel NaV1.7

Published:2021-05 Issue:9 Volume:109 Page:1497-1512.e6
ISSN:0896-6273
Container-title:Neuron
language:en
Short-container-title:Neuron

Author:

MacDonald Donald Iain,Sikandar Shafaq,Weiss Jan,Pyrski Martina,Luiz Ana P.,Millet Queensta,Emery Edward C.,Mancini Flavia,Iannetti Gian D.,Alles Sascha R.A.,Arcangeletti Manuel,Zhao Jing,Cox James J.,Brownstone Robert M.,Zufall Frank,Wood John N.

Funder

Versus Arthritis

Wellcome Trust

Publisher

Elsevier BV

Subject

General Neuroscience

Reference65 articles.

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2. Subtype-selective small molecule inhibitors reveal a fundamental role for Nav1.7 in nociceptor electrogenesis, axonal conduction and presynaptic release;Alexandrou;PLoS ONE,2016

3. Sensory neuron-derived NaV1.7 contributes to dorsal horn neuron excitability;Alles;Sci. Adv.,2020

4. Expression of Nav1.7 in DRG neurons extends from peripheral terminals in the skin to central preterminal branches and terminals in the dorsal horn;Black;Mol. Pain,2012

5. Organization and plasticity of sodium channel expression in the mouse olfactory and vomeronasal epithelia;Bolz;Front. Neuroanat.,2017

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