Targeting 14-3-3θ-mediated TDP-43 pathology in amyotrophic lateral sclerosis and frontotemporal dementia mice-Reference-Cited by-同舟云学术

Targeting 14-3-3θ-mediated TDP-43 pathology in amyotrophic lateral sclerosis and frontotemporal dementia mice

Published:2024-04 Issue:8 Volume:112 Page:1249-1264.e8
ISSN:0896-6273
Container-title:Neuron
language:en
Short-container-title:Neuron

Author:

Ke Yazi D.,van Hummel Annika^ORCID,Au Carol,Chan Gabriella,Lee Wei Siang,van der Hoven Julia^ORCID,Przybyla Magdalena^ORCID,Deng Yuanyuan^ORCID,Sabale Miheer,Morey Nicolle,Bertz Josefine^ORCID,Feiten Astrid^ORCID,Ippati Stefania^ORCID,Stevens Claire H.,Yang Shu,Gladbach Amadeus,Haass Nikolas K.^ORCID,Kril Jillian J.^ORCID,Blair Ian P.,Delerue Fabien,Ittner Lars M.^ORCID

Funder

MND Research Australia

National Health and Medical Research Council

Australian Research Council

National Institutes of Health

University of Queensland

National Institute on Alcohol Abuse and Alcoholism

Publisher

Elsevier BV

Reference73 articles.

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4. Partial loss of TDP-43 function causes phenotypes of amyotrophic lateral sclerosis;Yang;Proc. Natl. Acad. Sci. USA,2014

5. Loss of TDP-43 function underlies hippocampal and cortical synaptic deficits in TDP-43 proteinopathies;Ni;Mol. Psychiatry,2023

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. ALS-associated C21ORF2 variant disrupts DNA damage repair, mitochondrial metabolism, neuronal excitability and NEK1 levels in human motor neurons;Acta Neuropathologica Communications;2024-09-04

2. 14-3-3θ, a novel player in TDP-43 pathophysiology: Implications for ALS/FTD;Neuron;2024-04