Abrogating Native α-Synuclein Tetramers in Mice Causes a L-DOPA-Responsive Motor Syndrome Closely Resembling Parkinson’s Disease-Reference-Cited by-同舟云学术

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Abrogating Native α-Synuclein Tetramers in Mice Causes a L-DOPA-Responsive Motor Syndrome Closely Resembling Parkinson’s Disease

Published:2018-10 Issue:1 Volume:100 Page:75-90.e5
ISSN:0896-6273
Container-title:Neuron
language:en
Short-container-title:Neuron

Author:

Nuber Silke,Rajsombath Molly,Minakaki Georgia,Winkler Jürgen,Müller Christian P.,Ericsson Maria,Caldarone Barbara,Dettmer Ulf,Selkoe Dennis J.

Funder

NIH

Harvard NeuroDiscovery Center

Publisher

Elsevier BV

Subject

General Neuroscience

Reference70 articles.

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2. Phosphorylation of Ser-129 is the dominant pathological modification of alpha-synuclein in familial and sporadic Lewy body disease;Anderson;J. Biol. Chem.,2006

3. Alpha-synuclein p.H50Q, a novel pathogenic mutation for Parkinson’s disease;Appel-Cresswell;Mov. Disord.,2013

4. α-Synuclein occurs physiologically as a helically folded tetramer that resists aggregation;Bartels;Nature,2011

5. Sex hormones affect neurotransmitters and shape the adult female brain during hormonal transition periods;Barth;Front. Neurosci.,2015

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