IL-33/ST2 Signaling in the Pathogenesis of Chronic Pain and Itch-Reference-Cited by-同舟云学术

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IL-33/ST2 Signaling in the Pathogenesis of Chronic Pain and Itch

Published:2023-10 Issue: Volume:529 Page:16-22
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Gao Tian-Chi,Wang Cheng-Hao,Wang Yan-Qing,Mi Wen-Li^ORCID

Publisher

Elsevier BV

Subject

General Neuroscience

Reference51 articles.

1. Nociceptor interleukin 33 receptor/ST2 signaling in vibration-induced muscle pain in the rat;Alvarez;J Pain,2020

2. Interleukin-33 attenuates sepsis by enhancing neutrophil influx to the site of infection;Alves-Filho;Nat Med,2010

3. NKT cells are required to induce high IL-33 expression in hepatocytes during ConA-induced acute hepatitis;Arshad;Eur J Immunol,2011

4. Conjunctivitis: a systematic review of diagnosis and treatment;Azari;J Am Med Assoc,2013

5. Molecular characterization of NF-HEV, a nuclear factor preferentially expressed in human high endothelial venules;Baekkevold;Am J Pathol,2003

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Transient receptor potential melastatin 8 contributes to the interleukin-33-mediated cold allodynia in a mouse model of neuropathic pain;Pain;2024-08-08

2. Lewy Body Formation through the lens of Cytokine-Mediated Neuroinflammation in Parkinson's Disease Development;2024-04-08

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