The case for 8,5′-cyclopurine-2′-deoxynucleosides as endogenous DNA lesions that cause neurodegeneration in xeroderma pigmentosum-Reference-Cited by-同舟云学术

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The case for 8,5′-cyclopurine-2′-deoxynucleosides as endogenous DNA lesions that cause neurodegeneration in xeroderma pigmentosum

Published:2007-04 Issue:4 Volume:145 Page:1407-1417
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Brooks P.J.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference80 articles.

1. Xeroderma pigmentosum neurological abnormalities correlate with the colony forming ability after ultraviolet irradiation;Andrews;Proc Natl Acad Sci U S A,1978

2. The oxidative DNA lesion 8,5′-(S)-cyclo-2′-deoxyadenosine is repaired by the nucleotide excision repair pathway and blocks gene expression in mammalian cells;Brooks;J Biol Chem,2000

3. Cockayne syndrome;Brumback,2004

4. Genotoxic lipid peroxidation products: their DNA damaging properties and role in formation of endogenous DNA adducts;Burcham;Mutagenesis,1998

5. Mitochondrial free radical generation, oxidative stress, and aging;Cadenas;Free Radic Biol Med,2000

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