Bisphenol A depresses monosynaptic and polysynaptic reflexes in neonatal rat spinal cord in vitro involving estrogen receptor-dependent NO-mediated mechanisms

Author:

Pandey A.K.,Deshpande S.B.

Funder

UGC

Publisher

Elsevier BV

Subject

General Neuroscience

Reference44 articles.

1. Pancreatic insulin content regulation by the estrogen receptor ER alpha;Alonso-Magdalena;PLoS One,2008

2. Nitric oxide in the central nervous system: neuroprotection versus neurotoxicity;Calabrese;Nat Rev Neurosci,2007

3. Urinary concentrations of bisphenol A and 4-nonylphenol in a human reference population;Calafat;Environ Health Perspect,2005

4. Exposure of the U.S. population to Bisphenol A and 4-tertiary-octylphenol: 2003–2004;Calafat;Environ Health Perspect,2008

5. Prevention of phencyclidine-induced depression of the segmental reflex by l-3,4-dihydroxyphenylalanine in the rat spinal cord in vitro;Carp;J Pharmacol Exp Ther,1989

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