Neuroprotection of S-nitrosoglutathione against ischemic injury by down-regulating Fas S-nitrosylation and downstream signaling-Reference-Cited by-同舟云学术

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Neuroprotection of S-nitrosoglutathione against ischemic injury by down-regulating Fas S-nitrosylation and downstream signaling

Published:2013-09 Issue: Volume:248 Page:290-298
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Yin X.-H.,Yan J.-Z.,Hou X.-Y.,Wu S.-L.,Zhang G.-Y.

Funder

Jiangsu Higher Education Institutions

National Natural Science Foundation of China

Qing-Lan Project of Jiangsu Province

Publisher

Elsevier BV

Subject

General Neuroscience

Reference39 articles.

1. Treatment of ischemic brain damage by perturbing NMDA receptor-PSD-95 protein interactions;Aarts;Science,2002

2. Preconditioning regulation of bcl-2 and p66shc by human NOS1 enhances tolerance to oxidative stress;Andoh;FASEB J,2000

3. A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain;Boldin;J Biol Chem,1995

4. Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1 and TNF receptor-induced cell death;Boldin;Cell,1996

5. FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis;Chinnaiyan;Cell,1995

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1. Polarized benzene rings can promote the interaction between CaM and the CaMBD region of nNOS;Frontiers in Molecular Neuroscience;2024-09-03

2. Nitric Oxide-Dependent Pathways as Critical Factors in the Consequences and Recovery after Brain Ischemic Hypoxia;Biomolecules;2021-07-26

3. The Molecular Simulation Study of nNOS Activation Induced by the Interaction Between Its Calmodulin-Binding Domain and SUMO1;Frontiers in Molecular Neuroscience;2020-10-29

4. Protective Mechanism and Treatment of Neurogenesis in Cerebral Ischemia;Neurochemical Research;2020-08-13

5. Salidroside Ameliorates Mitochondria-Dependent Neuronal Apoptosis after Spinal Cord Ischemia-Reperfusion Injury Partially through Inhibiting Oxidative Stress and Promoting Mitophagy;Oxidative Medicine and Cellular Longevity;2020-07-23

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