Differential roles for Akt and mTORC1 in the hypertrophy of Pten mutant neurons, a cellular model of brain overgrowth disorders-Reference-Cited by-同舟云学术

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Differential roles for Akt and mTORC1 in the hypertrophy of Pten mutant neurons, a cellular model of brain overgrowth disorders

Published:2017-06 Issue: Volume:354 Page:196-207
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Nikolaeva Ina,Kazdoba Tatiana M.,Crowell Beth,D'Arcangelo Gabriella

Funder

New Jersey Commission on Brain Injury Research

by Exploration-Hypothesis Development

Department of Defense - CDMRP - TSCRP (G.D.)

Publisher

Elsevier BV

Subject

General Neuroscience

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2. An AKT3-FOXG1-reelin network underlies defective migration in human focal malformations of cortical development;Baek;Nat Med,2015

3. Loss of Tsc1 in vivo impairs hippocampal mGluR-LTD and increases excitatory synaptic function;Bateup;J Neurosci,2011

4. Insight into the structural mechanism for PKBalpha allosteric inhibition by molecular dynamics simulations and free energy calculations;Chen;J Mol Grap Model,2014

5. Evolving neurobiology of tuberous sclerosis complex;Crino;Acta Neuropathol,2013

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