Phosphorylation of 4.1N by CaMKII Regulates the Trafficking of GluA1-containing AMPA Receptors During Long-term Potentiation in Acute Rat Hippocampal Brain Slices-Reference-Cited by-同舟云学术

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Phosphorylation of 4.1N by CaMKII Regulates the Trafficking of GluA1-containing AMPA Receptors During Long-term Potentiation in Acute Rat Hippocampal Brain Slices

Published:2024-01 Issue: Volume:536 Page:131-142
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Yang Jun,Ma Rui-Ning,Dong Jia-Min,Hu Shu-Qun,Liu Yong^ORCID,Yan Jing-Zhi

Publisher

Elsevier BV

Subject

General Neuroscience

Reference58 articles.

1. LTP in hippocampal neurons is associated with a CaMKII-mediated increase in GluA1 surface expression;Appleby;J Neurochem,2011

2. Mechanisms of synaptic plasticity and recognition memory in the perirhinal cortex;Banks;Prog Mol Biol Transl Sci,2014

3. Modulation of AMPA receptor unitary conductance by synaptic activity;Benke;Nature,1998

4. Collapsin response mediator protein 2 (CRMP2) interacts with N-methyl-D-aspartate (NMDA) receptor and Na+/Ca2+ exchanger and regulates their functional activity;Brustovetsky;J Biol Chem,2014

5. SUMOylation and phosphorylation of GluK2 regulate kainate receptor trafficking and synaptic plasticity;Chamberlain;Nat Neurosci,2012

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