Presynaptic Dysfunction in Neurons Derived from Tay–Sachs iPSCs-Reference-Cited by-同舟云学术

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Presynaptic Dysfunction in Neurons Derived from Tay–Sachs iPSCs

Published:2019-08 Issue: Volume:414 Page:128-140
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Matsushita Kozo,Numakawa Tadahiro,Odaka Haruki,Kajihara Ryutaro,Soga Minami,Ozasa Shiro,Nakamura Kimitoshi,Mizuta Hiroshi,Era Takumi

Funder

Ministry of Health, Labor, and Welfare of Japan

Japan Agency for Medical Research and Development

Publisher

Elsevier BV

Subject

General Neuroscience

Reference39 articles.

1. Production of recombinant beta-hexosaminidase A, a potential enzyme for replacement therapy for Tay–Sachs and Sandhoff diseases, in the methylotrophic yeast Ogataea minuta;Akeboshi;Appl Environ Microbiol,2007

2. Activity-dependent fluorescent staining and destaining of living vertebrate motor nerve terminals;Betz;J Neurosci,1992

3. Neuron network activity scales exponentially with synapse density;Brewer;J Neural Eng,2009

4. Genetics and therapies for GM2 gangliosidosis;Cachon-Gonzalez;Curr Gene Ther,2018

5. Nestin mRNA expression correlates with the central nervous system progenitor cell state in many, but not all, regions of developing central nervous system;Dahlstrand;Brain Res Dev Brain Res,1995

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