Exocytosis is impaired in mucopolysaccharidosis IIIA mouse chromaffin cells-Reference-Cited by-同舟云学术

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Exocytosis is impaired in mucopolysaccharidosis IIIA mouse chromaffin cells

Published:2012-12 Issue: Volume:227 Page:110-118
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Keating D.J.,Winter M.A.,Hemsley K.M.,Mackenzie K.D.,Teo E.H.,Hopwood J.J.,Brooks D.A.,Parkinson-Lawrence E.J.

Funder

NHMRC Biomedical Training and the University of South Australia

Australian Research Council Future

Publisher

Elsevier BV

Subject

General Neuroscience

Reference36 articles.

1. Biogenesis of regulated exocytotic carriers in neuroendocrine cells;Benjamin;J Neurosci,2000

2. A novel missense mutation in lysosomal sulfamidase is the basis of MPS IIIA in a spontaneous mouse mutant;Bhattacharyya;Glycobiology,2001

3. A mouse model for mucopolysaccharidosis type IIIA (Sanfilippo syndrome);Bhaumik;Glycobiology,1999

4. Analysis of regulated exocytosis in adrenal chromaffin cells: insights into NSF/SNAP/SNARE function;Burgoyne;Bioassays,1998

5. Lipid dynamics in exocytosis;Chasserot-Golaz;Cell Mol Neurobiol,2010

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