Apoptosis Due to After-effects of Acute Ethanol Exposure in Brain Cortex: Intrinsic and Extrinsic Signaling Pathways-Reference-Cited by-同舟云学术

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Apoptosis Due to After-effects of Acute Ethanol Exposure in Brain Cortex: Intrinsic and Extrinsic Signaling Pathways

Published:2024-04 Issue: Volume:544 Page:39-49
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Karadayian Analía G.,Czerniczyniec Analia,Lores-Arnaiz Silvia

Funder

Consejo Nacional de Investigaciones Científicas y Técnicas

Universidad de Buenos Aires

Agencia Nacional de Promoción de la Investigación, el Desarrollo Tecnológico y la Innovación

Publisher

Elsevier BV

Reference53 articles.

1. Transient activation of microglia following acute alcohol exposure in developing mouse neocortex is primarily driven by BAX-dependent neurodegeneration;Ahlers;Glia,2015

2. Low citrate synthase activity is associated with glucose intolerance and lipotoxicity;Alhindi;J Nutr Metab,2019

3. PARP-1 inhibition increases mitochondrial metabolism through SIRT1 activation;Bai;Cell Metab,2011

4. Influence of chronic alcohol treatment on the expression of the Bdnf, Bax, Bcl-xL, and CASP3 genes in the mouse brain: Role of the C1473G polymorphism in the gene encoding tryptophan hydroxylase 2;Bazovkina;Mol Biol (Mosk),2016

5. Pharmacological regulation of mitochondrial nitric oxide synthase;Boveris;Methods Enzym,2002

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