Thyroid hormone is required for the pruning of afferent type II spiral ganglion neurons in the mouse cochlea
Author:
Funder
National Institute in Deafness and other Communicative Disorders
Publisher
Elsevier BV
Subject
General Neuroscience
Reference55 articles.
1. Type I vs type II spiral ganglion neurons exhibit differential survival and neuritogenesis during cochlear development;Barclay;Neural Dev,2011
2. Calcium- and otoferlin-dependent exocytosis by immature outer hair cells;Beurg;J Neurosci,2008
3. Thyroid hormone deficiency affects postnatal spiking activity and expression of Ca2+ and K+ channels in rodent inner hair cells;Brandt;J Neurosci,2007
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5. Type 2 iodothyronine deiodinase expression in the cochlea before the onset of hearing;Campos-Barros;Proc Natl Acad Sci U S A,2000
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1. Tissue engineering strategies for spiral ganglion neuron protection and regeneration;Journal of Nanobiotechnology;2024-07-31
2. Thyroid hormone controls the timing of cochlear ribbon synapse maturation;Biochemical and Biophysical Research Communications;2024-04
3. Macrophages Are Dispensable for Postnatal Pruning of the Cochlear Ribbon Synapses;Frontiers in Cellular Neuroscience;2021-10-21
4. Cochlear Fibrocyte and Osteoblast Lineages Expressing Type 2 Deiodinase Identified with a Dio2CreERt2 Allele;Endocrinology;2021-08-26
5. Association between SSNHL and Thyroid Diseases;International Journal of Environmental Research and Public Health;2020-11-13
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