An impaired cardiodynamic phase contributes to the abnormal vo2 kinetics at exercise onset in both congestive heart failure and heart transplant patients but results from differing mechanisms
Author:
Publisher
Elsevier BV
Subject
Transplantation,Surgery
Reference17 articles.
1. Value of peak exercise oxygen consumption for optimal timing of cardiac transplantation in ambulatory patients with heart failure.
2. Central and peripheral limitations to upright exercise in untrained cardiac transplant recipients.
3. Persistent Exercise Intolerance Following Cardiac Transplantation Despite Normal Oxygen Transport
4. Excessive oxygen deficit during low-level exercise in heart failure
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1. Priming cardiac function with voluntary respiratory maneuvers and effect on early exercise oxygen uptake;Journal of Applied Physiology;2022-05-01
2. Exercise on-transition uncoupling of ventilatory, gas exchange and cardiac hemodynamic kinetics accompany pulmonary oxygen stores depletion to impact exercise intolerance in human heart failure;Acta Physiologica;2018-04-01
3. Speeding of pulmonary VO2 on-kinetics by light-to-moderate-intensity aerobic exercise training in chronic heart failure: Clinical and pathophysiological correlates;International Journal of Cardiology;2013-09
4. Duration of “Phase I” V̇o2p: a comparison of methods used in its estimation and the effects of varying moderate-intensity work rate;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2013-02-01
5. Age-related prolongation of phase I of V̇o2 on-kinetics in healthy humans;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2010-09
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